![]() This means that psychosis becomes more severe with repeated intermittent use. Ĭocaine-induced psychosis shows sensitization toward the psychotic effects of the drug. Delusional parasitosis with formication ("cocaine bugs") is also a fairly common symptom. Typical symptoms of sufferers include paranoid delusions that they are being followed and that their drug use is being watched accompanied by hallucinations that support the delusional beliefs. CocaineĬocaine has a similar potential to induce temporary psychosis with more than half of cocaine abusers reporting at least some psychotic symptoms at some point. However, unlike similar disorders, in AWP, substituted amphetamines reduce rather than increase symptoms, and the psychosis or mania resolves with resumption of the previous dosing schedule. Īlthough rare and not formally recognized, a condition known as Amphetamine Withdrawal Psychosis (AWP) may occur upon cessation of substituted amphetamine use and, as the name implies, involves psychosis that appears on withdrawal from substituted amphetamines. ![]() The disorders are often distinguished by a rapid resolution of symptoms in amphetamine psychosis, while schizophrenia is more likely to follow a chronic course. Relatives of methamphetamine users with a history of amphetamine psychosis are five times more likely to have been diagnosed with schizophrenia than relatives of methamphetamine users without a history of amphetamine psychosis. There is some evidence that vulnerability to amphetamine psychosis and schizophrenia may be genetically related. Amphetamine psychosis may be purely related to high drug usage, or high drug usage may trigger an underlying vulnerability to schizophrenia. Symptoms of acute amphetamine psychosis are very similar to those of the acute phase of schizophrenia although in amphetamine psychosis visual hallucinations are more common and thought disorder is rare. Furthermore, even at a small dose, the psychosis can be quickly re-established. However, it has been suggested that around 5–15% of users fail to make a complete recovery in the long-term. A Japanese study of recovery from methamphetamine psychosis reported a 64% recovery rate within 10 days rising to an 82% recovery rate at 30 days after methamphetamine cessation. The symptoms of amphetamine psychosis include auditory and visuals, delusions of persecution and delusions of reference concurrent with both clear consciousness and prominent extreme agitation. Common examples include DOM, ephedrine, MDMA, and methamphetamine. The following stimulants are known to cause psychosis.ĭrugs in the class of amphetamines are known to induce "amphetamine psychosis" typically when chronically abused or used in high doses. ![]() These additional symptoms may include aggression, arrhythmia, dilated pupils, diarrhea, hypertension, hyperthermia, nausea, rapid breathing, restlessness, seizures, sleep deprivation, tremor, and vomiting. In cases of stimulant psychosis, not organic psychosis, patients tend to also present with the physical symptoms of prolonged stimulant abuse or acute overdose. The symptoms of stimulant psychosis may vary slightly depending on the drug ingested but generally include the symptoms of organic psychosis including external hallucinations, delusions, thought disorder, environmental alterations, object activation, and, in extreme cases, autonomous entities such as shadow people and catatonia.
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